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    INTERNATIONAL PROGRAMME ON CHEMICAL SAFETY

    WORLD HEALTH ORGANIZATION



    TOXICOLOGICAL EVALUATION OF SOME
    FOOD COLOURS, ENZYMES, FLAVOUR
    ENHANCERS, THICKENING AGENTS, AND
    CERTAIN FOOD ADDITIVES



    WHO FOOD ADDITIVES SERIES 6







    The evaluations contained in this publication were prepared by the
    Joint FAO/WHO Expert Committee on Food Additives which met in Rome,
    4-13 June 19741


    World Health Organization     Geneva     1975






              

    1  Eighteenth Report of the Joint FAO/WHO Expert Committee on
    Food Additives, Wld Hlth Org. techn. Rep. Ser., 1974, No. 557.
    FAO Nutrition Meetings Report Series, 1974, No. 54.

    AZORUBINE

    BIOLOGICAL DATA

    BIOCHEMICAL ASPECTS

         Rats were injected intravenously with approximately 1 mg of the
    dye. The bile was collected for six hours and analysed. The recovery
    of the dye was an average of 38% (30-40%) of the administered quantity
    (Ryan & Wright, 1961). This like any other azo dye is probably reduced
    in the gut by bacterial azo reductases (Walker, 1970).

    TOXICOLOGICAL STUDIES

    Special studies on Heinz bodies

    Cat

         Four cats were given 5% aqueous solution in doses of 1g on the
    first day and 0.1 g on the ninth and eighteenth day. A negative test
    for Heinz bodies was obtained (Deutsche Forsch., 1957).

    Special studies on mutagenicity

         The colour was tested for mutagenic action in a concentration of
    0.5 g/100 ml in cultures of Escherichia coli. No mutagenic effect
    was found (Lück & Rickerl, 1960).

    Special studies on reproduction

    Rat

         Twenty-five male and 25 female rats received 1% azorubine in
    their drinking water for 180 days, giving approximately 55 g dye per
    animal. A similar group of 50 rats acted as controls. Weight gain,
    mortality and general condition were similar in both groups. After
    seven months animals were mated and an F1 generation produced. After
    weaning the pups were put again on 1% azorubine and after four months
    mated to produce an F2 generation. No abnormalities regarding litter
    number or fertility were noted. After 200 days on 1% azorubine the F2
    generation was kept on normal diet and water for two years. No adverse
    effects were seen on mortality or tumour incidence (Hecht, 1966).

    Special studies on sensitizing effects

    Guinea-pig

         In experiments on guinea-pigs, it was found that this colour had
    no sensitizing activity (Bär & Griepentrog, 1960).

    Acute toxicity
                                                                        

                             LD50
    Animal       Route       (mg/kg bw)          Reference
                                                                        

    Mouse        oral             1 000          Deutsche Forsch., 1957
                 oral             8 000          Gaunt et al., 1967
                 i.p.               900          Gaunt et al., 1967

                 i.v.               800          Deutsche Forsch., 1957

    Rat          oral            10 000          Gaunt et al., 1967
                 oral             1 000          Deutsche Forsch., 1957
                 i.p.             1 000          Deutsche Forsch., 1957
                 i.p.         900-1 100          Gaunt et al., 1967
                 i.v.               800          Deutsche Forsch., 1957
                                                                        

    Short-term studies

    Rat

         Three weanling rats were given a 0.1% solution of carmoisine to
    drink for 28 days (daily consumption approximately 15 mg). No toxic
    effects were noted (Goldblatt & Frodsham, 1952).

         Five groups of 16 male and 16 female rats were fed diets
    containing 0, 0.05, 0.1, 0.5 and 1.0% carmoisine for three months.
    There were no adverse effects on general health, growth or food
    consumption and no departure from normality was seen in the
    haematological studies and in the liver and kidney function tests. The
    principal organ weight finding was an elevated relative kidney weight
    at 1% in females. The types and incidence of histological changes were
    comparable in control and test groups (Gaunt et al., 1967).

    Pig

         Four groups of three male and three female pigs received
    carmoisine at dose levels of 0, 250, 500 or 1000 mg/kg/day for three
    months. There were no adverse effects on growth and no differences
    between treated and control groups were evident in the haematological
    examination, serum chemistry, urine analyses, organ weights or
    histological examination (Gaunt et al., 1969).

    Long-term studies

    Mouse

         Five groups of 30 male and 30 female mice were fed diets
    containing 0, 300, 900, 2700 or 8100 ppm of the colour for 80 weeks. A
    high death rate was noted in all test groups and in the controls. A
    further test was therefore undertaken with the same number of mice but
    using dosage levels of 0, 100, 500, 2500 and 12 500 ppm. There were no
    adverse effects on mortality, weight gain, organ weights or incidence
    of histopathological findings including tumours. There was a mild
    anaemia in mice given the highest level (BIBRA, 1973).

         A group of 30 mice, comprising equal numbers of both sexes were
    given 0.1 ml of a 3% suspension of the colour in arachis oil; after
    six months the strength of the suspension was increased to 6%. The
    colour was given subcutaneously for 360 days. After this period the
    animals were kept for 630 days. The weekly dose was respectively 6 and
    12 mg and the total dose was 468 mg/mouse. Seven lymphomas and one
    intestinal tumour were observed. In 60 control animals four lymphomas
    and an intestinal tumour were present (Bonser et al., 1956).

    Rat

         Ten rats were given the colour in the drinking water in a
    concentration of 1% for 209 days. The daily intake was 1.2 g/kg bw and
    the total amount administered was 51 g per animal. The observation
    period was 919 days. No tumours were found (Deutsche Forsch., 1957).

         Ten rats were given 1% of the colour in the drinking water for
    250 days. The daily intake was 7.94 g/kg bw and the total intake
    52 g/animal. The observation period was 545 days. No tumours were
    found (Deutsche Forsch., 1947).

         Ten rats were given a diet containing 0.2% of the colour for 417
    days. The daily intake was approximately 0.1 g/kg bw and the total
    intake was 11 g per animal. The observation period was 838 days. No
    tumours were found (Deutsche Forsch., 1957).

         A group of 10 rats was given twice weekly subcutaneous injections
    of 0.5 ml of a 1% solution (= 5 mg) of the colour for one year. The
    animals were kept under observation for over 938 days. One axillary
    tumour was observed in one animal (Deutsche Forsch., 1957).

         In a repeat experiment another group of 10 rats was given twice
    weekly subcutaneously 0.5 ml of a 1% solution (= 5 mg) of the colour
    for one year. No tumours were formed after 521 days each animal having
    received 0.5 g (Deutsche Forsch., 1957).

    Comments:

         Little information is available on the metabolism of this colour.
    It has been studied adequately long-term only in the mouse. The long-
    term studies in the rat recorded only tumour incidence and survival
    while many other essential observations have not been reported. The
    reproduction study did not reveal any compound-related adverse
    effects. There are no studies available on embryotoxicity including
    teratology.

    EVALUATION

    Level causing no toxicological effect

         Rat: 0.2% (= 2000 ppm) in the diet equivalent to 100 mg/kg bw.

    Estimate of acceptable daily intake for man

         0-0.5 mg/kg bw*

    FURTHER WORK OR INFORMATION

         Required by June 1978

         (a)     adequate long-term studies in another species;

         (b)     investigation of embryotoxicity including teratogenicity;

         (c)     adequate metabolic studies in several species preferably
         including man.

    REFERENCES

    Bär, F. & Griepentrog, F. (1960) Med. u. Ernährung, 1, 99

    BIBRA (1973) Research Report No. 8/1973


    Bonser, G. M., Clayson, D. B. & Jull, J. W. (1956) Brit. J. Cancer,
         10, 653

    Deutsche Forschungsgemeinschaft, Bad Godesberg, Federal Republic of
         Germany, Farbstoff Kommission (1957) Mitteilung 6

    Gaunt, I. F. et al. (1967) Fd. Cosmet Toxicol., 5, 179

    Gaunt, I. F. et al. (1969) Fd. Cosmet Toxicol., 7, 1

              

    *    Temporary.

    Lück, H. & Rickerl, E. (1960) Z. Lebensmitt.-Untersuch., 112, 157

    Ryan, A. J. & Wright, S. E. (1961) J. Pharm. Pharmacol., 13, 493

    Walker, R. (1970) Fd. Cosmet Toxicol., 8, 659

    Goldblatt & Frodsham (1952) Unpublished information from ICI

    Hecht, G. (1966) Report to WHO


    See Also:
       Toxicological Abbreviations
       Azorubine (WHO Food Additives Series 13)
       Azorubine (WHO Food Additives Series 18)
       AZORUBINE (JECFA Evaluation)